effe sleur en pleur van mijn eigen posts maar van ee andere site (ben te lui om te vertalen)
About DOMS :
THIS is what professionals say :
"
Subsequent research has suggested that damage to the muscle ultra structure and connective tissue may be responsible for DOMS. It is suggested that a sequence of events starting with exercise causes muscle damage and then muscle protein breakdown, resulting in cell inflammation and increased local muscle temperature. As a result pain receptors are activated, causing the sensation of DOMS."
http://www.sportsinjurybulletin.com/archive/1077-muscle-soreness.htm
"
DOMS is thought to be a result of microscopic tearing of the muscle fibers."
http://www.physsportsmed.com/issues/1999/01_99/muscle.htm
THIS is what people make of it :
"
DOMS occurs by the following mechanism: Strenuous exercise leads to structural damage to muscle cells which causes calcium to leak out of the muscle. This causes the activation of enzymes that breaks down cellular proteins in the muscle. These proteins then cause an inflammatory response by the immune system which then leads to edema (water retention at the site of injury) and pain."
So, what do i want to say ? BE CAREFULL WHEN YOU READ SOMETHING
all too often "
is suggested that" turns into "
is". THAT IS NOT CORRECT !!!
just because something is suggested, it doesn't mean it's true for a 100% !!!
just to show others who 'claim' that muscle soreness is indeed caused by these little damages...
Delayed-onset muscle soreness does not reflect the magnitude of eccentric exercise-induced muscle damage.
Nosaka K, Newton M, Sacco P.
Exercise and Sports Science, Graduate School of Integrated Science, Yokohama City University, Yokohama, Japan, School of Biomedical and Sports Science, Edith Cowan University, Western Australia, Australia.
This study investigated the relationship between delayed-onset muscle soreness and other indicators of muscle damage following eccentric exercise. Male students (n = 110) performed 12 (12ECC), 24 (24ECC), or 60 maximal eccentric actions of the elbow flexors (60ECC). Maximal isometric force, relaxed and flexed elbow joint angles, upper arm circumference, and plasma creatine kinase activity were assessed immediately before and after, and for 4 days after exercise. Muscle soreness (SOR) was evaluated by a visual analog scale (a 50-mm line, 0: no pain, 50: extremely painful) when the elbow flexors were palpated (SOR-Pal), flexed (SOR-Flx) and stretched (SOR-Ext). Although 24ECC and 60ECC resulted in significantly (P <; 0.05) larger changes in all indicators and slower recovery compared to 12ECC, no significant differences were evident for SOR-Pal and SOR-Flx between 12ECC and 24ECC, or 12ECC and 60ECC. In contrast, SOR-Ext was significantly (P <; 0.05) lower for 12ECC compared to 24ECC and 60ECC. A Pearson product-moment correlation showed SOR-Pal did not correlate significantly with any indicators, however, SOR-Ext and SOR-Flx showed weak (r <; 0.32) but significant (P <; 0.05) correlations with other indicators. Because of generally poor correlations between DOMS and other indicators, we conclude
that use of DOMS is a poor reflector of eccentric exercise-induced muscle damage and inflammation, and changes in indirect markers of muscle damage and inflammation are not necessarily accompanied with DOMS.
PMID: 12453160
hope i passed the message