vitaminen

[remy187]

Welke vitaminen nemen jullie als je aan het kuren bent, en hoeveel?
Grtz

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[Big-T]

Belangrijk is B reeks, C en mineralen.

B helpt de lever te zuiveren en gaat puistjes tegen, bevorderd verder nog een aatal spier gerelateerde functies en de stofwisseling.

C is belanrijk voor het immuumsysteem zodat je niet ziek wordt tijdens de kuur.

Mineralen zink, magnesium ed zijne extra nodig door de grotere spierbelasting en snellere groei.

Verder moet je niet teveel vitaminen nemen want sommige zijn ook extra belastend voor de organen en grootste deel komt wel uit voeding.

 

[bartp]

Vitamen E moest je mijden ivm blokeren receptoren geloof ik ooit gelezen te hebben. Kan iemand vertellen of dit klopt en zo ja waarom dit zo is?

 

[Big-T]

Goed punt, denk dat je het misschien in de elite sectie hebt gelezen

VERMIJD ten alle koste vitamine E, het vermindert het aantal androgene receptoren.

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Waarom vit E dat doet weet ik niet precies maar de bigc zal wel gelijk hebben, het kwam uit een uitgebreide research van hem voor een kuur.

 

[Big-T]

Gevonden:

Mol Cancer Ther. 2003 Aug;2(8):797-803. Related Articles, Links


Androgen antagonist activity by the antioxidant moiety of vitamin E, 2,2,5,7,8-pentamethyl-6-chromanol in human prostate carcinoma cells.

Thompson TA, Wilding G.

University of Wisconsin Comprehensive Cancer Center, University of Wisconsin-Madison, Madison, Wisconsin 53792, USA.

Antioxidants, such as vitamin E, are being investigated for efficacy in prostate cancer prevention. In this study, we show that the antioxidant moiety of vitamin E, 2,2,5,7,8-pentamethyl-6-chromanol (PMCol), has antiandrogen activity in prostate carcinoma cells. In the presence of PMCol, the androgen-stimulated biphasic growth curve of LNCaP human prostate carcinoma cells was shifted to the right. The PMCol-induced growth shift was similar to that produced by treatment with the pure antiandrogen bicalutamide (i.e., Casodex), indicative of androgen receptor (AR) antagonist activity. The concentration of PMCol used was below the concentration required to affect cell growth or viability in the absence of androgen. Using an AR binding competition assay, PMCol was found to be a potent antiandrogen in both LNCaP and LAPC4 cells, with an IC(50) of approximately 10 micro M against 1 nM R1881 (methyltrienolone; a stable, synthetic androgen). Prostate-specific antigen release from LNCaP cells produced by androgen exposure with either 0.05 or 1.0 nM R1881 was inhibited 100% and 80%, respectively, by 30 micro M PMCol. Also, PMCol inhibited androgen-induced promoter activation in both LNCaP and LAPC4 cells. However, PMCol did not affect AR protein levels, suggesting that the inhibitory effects of PMCol on androgenic pathways were not due to decreased expression of the AR. Therefore, growth modulation by the antioxidant moiety of vitamin E in androgen-sensitive prostate carcinoma cells is due, at least in part, to its potent antiandrogenic activity.

PMID: 12939470 [PubMed - indexed for MEDLINE]

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Vitamin E succinate inhibits the function of androgen receptor and the expression of prostate-specific antigen in prostate cancer cells.

Zhang Y, Ni J, Messing EM, Chang E, Yang CR, Yeh S.

Department of Urology, University of Rochester, Rochester, NY 14642, USA.

Although epidemiological evidence indicates that a daily supplement of vitamin E may reduce the risk of prostate cancer, the detailed mechanism underlying this effect remains unclear. Here we demonstrate that alpha-tocopheryl succinate (VES) can suppress the expression of prostate-specific antigen (PSA), a marker for the progression of prostate cancer. VES can also suppress androgen receptor (AR) expression by means of transcriptional and posttranscriptional modulation, but not ligand binding, nuclear translocation, or AR dimerization. This VES-mediated inhibition of AR is selective because VES does not repress the expression of other nuclear receptors. Cell growth studies further show that VES inhibits the growth of prostate cancer LNCaP cells. In contrast, hydroxyflutamide (HF), an antiandrogen currently used to treat prostate cancer patients, only slightly inhibits LNCaP cell growth. Interestingly, simultaneous addition of HF and VES results in a more significant inhibition of LNCaP cell growth. Moreover, selenomethionine (SM), a prostate cancer treatment adjuvant, shows an inhibitory effect on LNCaP cell growth, yet has no effect on the AR/PSA pathway. Together, our data indicate that VES may suppress androgen/AR-mediated cell growth and PSA expression by inhibiting AR expression at both the transcription and translation levels. This previously undescribed mechanism may explain how VES inhibits the growth of prostate cancer cells and help us to establish new therapeutic concepts for the prevention and treatment of prostate cancer.

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Goed tegen prostaatkanker maar minder voor spiergroei en kracht dus...

 

[ECLIPSE]

ik neem b5 en c, zinc en magnesium tijdens de kuur.. en een sterke multi na de kuur en tijdens de kuur als ik me wat ziekjes voel

 

[remy187]

Hey bedankt voor alle info..
Sta er van te kijken dat iedereen hier echt moeite doet om elkaar te helpen..
Grtzz.

 

[2hard2die]

Ik neem altijd vit c1 1000mg per dag (wel time-released!!!)
en een multivit tab per dag