Was vandaag toevallig in een tent in Amsterdam waar ze het hadden opgesplits... één gedeelte rokers andere gedeelte niet rokers.. en zoals Roxxe zei.. gewoon de ondernemers laten beslissen... zo kunnen de klanten ook beslissen of ze naar een rokers horeca onderneming gaan of naar een niet rokers onderneming.
Passief roken veroorzaakt GEEN longkanker
- Topic starter roxxe
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Als zoiets in de praktijk werkt juich ik dat alleen maar toe, maar tot nu toe zie je het bijna nergens helaas.
Werkt perfect in de praktijk.. en vandaag was ik in L'Opera in Amsterdam en daar was het gesplits.. ik zat gewoon m'n broodje op te eten en m'n choco te drinken in een rrokvrije zone terwijl aan de andere kant wat feestende mensen hun sigaretje zaten te roken... leven en laten leven zeg ik altijd..
Als er de keus is waarom zou het niet werken in de praktijk? gaat om vraag en aanbod...
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voila iederen tevree 
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Big'r
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Laten we onszelf gewoon eens bij de feiten houden. Wat dat betreft vertrouw ik sterke wetenschappelijke bewijslast boven de mening van wie dan ook... En helaas roxxe, meeroken is wel schadelijk.
PASSIEF MEEROKEN VERHOOGT NIET ALLEEN DE KANS OP LONGKANKER:
Air pollution and lung cancer risks in China--a meta-analysis.
Lung cancer is a serious health problem in China, as in the rest of the world. Many studies have already proved that air pollution as well as other environmental factors can increase the risk of lung cancer. Based on epidemiological studies carried out in China, this paper proposes odds ratios (OR) to evaluate the risk of lung cancer from indoor air pollution for the Chinese population by applying the method of meta-analysis. For domestic coal use for heating and cooking, the pooled OR values are 1.83 (95% CI: 0.62-5.41) and 2.66 (1.39-5.07) for women and both coïtuses, respectively. For indoor exposure to coal dust, the OR values are 2.52 (95% CI: 1.94-3.28) and 2.42 (1.62-3.63) for women and both coïtuses, respectively. Cooking oil vapor is another factor increasing lung cancer risk. The OR values are 2.12 (95%CI: 1.81-2.47), 1.78 (1.50-2.12) and 6.20 (2.88-13.32) for nonsmoking women, women, and both coïtuses, respectively. Regarding environmental tobacco smoke, the pooled OR values are 1.70 (95% CI: 1.32-2.18) and 1.64 (1.29-2.07) for nonsmoking women and both coïtuses, respectively. Funnel plots with statistical test have been applied to examine the publication bias, and the results implied that the analysis of coal consumption and cooking oil pollution might be affected by publication bias. The meta-analysis results confirm the association between lung cancer and indoor air pollution for the Chinese population.
Revisiting evidence on lung cancer and passive smoking: adjustment for publication bias by means of "trim and fill" algorithm.
Meta-analyses are subject to bias because smaller or non-significant studies are less likely to be published, and most meta-analyses do not consider the effect of publication bias on their results. To assess the true risk, we revisited a famous meta-analysis including 37 studies on lung cancer and passive smoking, and adjusted for publication bias by means of the "trim and fill" algorithm. The adjusted relative risk of lung cancer in non-smokers who lived with a smoker from the 44 studies including the 7 filled ones was 1.19 (95% confidence interval 1.08-1.31, p = 0.0004), and the estimate of excess risk fell from 24 to 19%.
MAAR OOK DE KANS OM TE OVERLIJDEN BIJ HARTZIEKTEN:
The impact of environmental tobacco smoke on women's risk of dying from heart disease: a meta-analysis.
OBJECTIVE: To review systematically and analyze the association between environmental tobacco smoke (ETS) exposure and the risk of dying from heart disease in women. METHODS: We searched the English-language literature using MEDLINE (1966-April 2004), CINAHL, PsychInfo, and bibliographies of selected studies. We included studies that specifically addressed the association of ETS and heart disease mortality in women and had adequate controls and retrievable risk estimates. We looked for either cohort studies or randomized controlled trials. Studies were evaluated independently by two of the authors. Nine cohort studies were finally selected for analysis. We estimated the summary relative risk (RR) and associated 95% confidence intervals (95% CI) using a random-effects model. RESULTS: Mean follow-up periods for these cohorts ranged from 6 to 39 years. Among non-smoking women, exposure to ETS was associated with a 15% increase in the risk of dying from heart disease compared with nonsmoking women not exposed to ETS (RR = 1.15, 95% CI 1.03-1.28, p < 0.05). CONCLUSIONS: Among nonsmoking women, exposure to passive smoke increases the risk of dying from heart disease. In accordance with the newly developed guidelines by the American Heart Association for prevention of cardiovascular disease (CVD) in women, we recommend counseling women on reducing or avoiding ETS exposure.
Passive smoking and coronary heart disease.
A large series of clinico-epidemiological studies, meta-analyses and experimental findings have concluded that there is a relationship between coronary heart disease (CHD) and passive smoking either after acute or chronic exposure. Cigarette smoking is the most important cause of premature death in industrialized countries because it is associated with an increased risk of developing several types of cancer and arterial disease. In family homes as well as in workplaces, environmental tobacco smoke (ETS) exposure is associated with an increased risk of CHD in exposed non-smokers when compared to un-exposed non-smokers. Different anatomical structures are damaged by ETS. The endothelium, artery wall and heart are target organs for passive smoking. Therefore, smoking cessation will benefit both smokers and those exposed to ETS.
NIET BIJ IEDEREEN BEKEND IS DAT ACTIEF ROKEN NIET ALLEEN DE KANS OP LONGKANKER VERHOOGT, MAAR OOK DIE VAN EEN BREED SCALA AAN ANDERE KANKERSOORTEN:
Tobacco smoking and cancer: a brief review of recent epidemiological evidence.
This report summarises the epidemiological evidence on the association between tobacco smoking and cancer, which was reviewed by an international group of scientists convened by IARC. Studies published since the 1986 IARC Monograph on "Tobacco smoking" provide sufficient evidence to establish a causal association between cigarette smoking and cancer of the nasal cavities and paranasal sinuses, nasopharynx, stomach, liver, kidney (renal cell carcinoma) and uterine cervix, and for adenocarcinoma of the oesophagus and myeloid leukaemia. These sites add to the previously established list of cancers causally associated with cigarette smoking, namely cancer of the lung, oral cavity, pharynx, larynx, oesophagus, pancreas, urinary bladder and renal pelvis. Other forms of tobacco smoking, such as cigars, pipes and bidis, also increase risk for cancer, including cancer of the lung and parts of the upper aerodigestive tract. A meta-analysis of over 50 studies on involuntary smoking among never smokers showed a consistent and statistically significant association between exposure to environmental tobacco smoke and lung cancer risk. Smoking is currently responsible for a third of all cancer deaths in many Western countries. It has been estimated that every other smoker will be killed by tobacco.
PASSIEF MEEROKEN VERHOOGT NIET ALLEEN DE KANS OP LONGKANKER:
Air pollution and lung cancer risks in China--a meta-analysis.
Lung cancer is a serious health problem in China, as in the rest of the world. Many studies have already proved that air pollution as well as other environmental factors can increase the risk of lung cancer. Based on epidemiological studies carried out in China, this paper proposes odds ratios (OR) to evaluate the risk of lung cancer from indoor air pollution for the Chinese population by applying the method of meta-analysis. For domestic coal use for heating and cooking, the pooled OR values are 1.83 (95% CI: 0.62-5.41) and 2.66 (1.39-5.07) for women and both coïtuses, respectively. For indoor exposure to coal dust, the OR values are 2.52 (95% CI: 1.94-3.28) and 2.42 (1.62-3.63) for women and both coïtuses, respectively. Cooking oil vapor is another factor increasing lung cancer risk. The OR values are 2.12 (95%CI: 1.81-2.47), 1.78 (1.50-2.12) and 6.20 (2.88-13.32) for nonsmoking women, women, and both coïtuses, respectively. Regarding environmental tobacco smoke, the pooled OR values are 1.70 (95% CI: 1.32-2.18) and 1.64 (1.29-2.07) for nonsmoking women and both coïtuses, respectively. Funnel plots with statistical test have been applied to examine the publication bias, and the results implied that the analysis of coal consumption and cooking oil pollution might be affected by publication bias. The meta-analysis results confirm the association between lung cancer and indoor air pollution for the Chinese population.
Revisiting evidence on lung cancer and passive smoking: adjustment for publication bias by means of "trim and fill" algorithm.
Meta-analyses are subject to bias because smaller or non-significant studies are less likely to be published, and most meta-analyses do not consider the effect of publication bias on their results. To assess the true risk, we revisited a famous meta-analysis including 37 studies on lung cancer and passive smoking, and adjusted for publication bias by means of the "trim and fill" algorithm. The adjusted relative risk of lung cancer in non-smokers who lived with a smoker from the 44 studies including the 7 filled ones was 1.19 (95% confidence interval 1.08-1.31, p = 0.0004), and the estimate of excess risk fell from 24 to 19%.
MAAR OOK DE KANS OM TE OVERLIJDEN BIJ HARTZIEKTEN:
The impact of environmental tobacco smoke on women's risk of dying from heart disease: a meta-analysis.
OBJECTIVE: To review systematically and analyze the association between environmental tobacco smoke (ETS) exposure and the risk of dying from heart disease in women. METHODS: We searched the English-language literature using MEDLINE (1966-April 2004), CINAHL, PsychInfo, and bibliographies of selected studies. We included studies that specifically addressed the association of ETS and heart disease mortality in women and had adequate controls and retrievable risk estimates. We looked for either cohort studies or randomized controlled trials. Studies were evaluated independently by two of the authors. Nine cohort studies were finally selected for analysis. We estimated the summary relative risk (RR) and associated 95% confidence intervals (95% CI) using a random-effects model. RESULTS: Mean follow-up periods for these cohorts ranged from 6 to 39 years. Among non-smoking women, exposure to ETS was associated with a 15% increase in the risk of dying from heart disease compared with nonsmoking women not exposed to ETS (RR = 1.15, 95% CI 1.03-1.28, p < 0.05). CONCLUSIONS: Among nonsmoking women, exposure to passive smoke increases the risk of dying from heart disease. In accordance with the newly developed guidelines by the American Heart Association for prevention of cardiovascular disease (CVD) in women, we recommend counseling women on reducing or avoiding ETS exposure.
Passive smoking and coronary heart disease.
A large series of clinico-epidemiological studies, meta-analyses and experimental findings have concluded that there is a relationship between coronary heart disease (CHD) and passive smoking either after acute or chronic exposure. Cigarette smoking is the most important cause of premature death in industrialized countries because it is associated with an increased risk of developing several types of cancer and arterial disease. In family homes as well as in workplaces, environmental tobacco smoke (ETS) exposure is associated with an increased risk of CHD in exposed non-smokers when compared to un-exposed non-smokers. Different anatomical structures are damaged by ETS. The endothelium, artery wall and heart are target organs for passive smoking. Therefore, smoking cessation will benefit both smokers and those exposed to ETS.
NIET BIJ IEDEREEN BEKEND IS DAT ACTIEF ROKEN NIET ALLEEN DE KANS OP LONGKANKER VERHOOGT, MAAR OOK DIE VAN EEN BREED SCALA AAN ANDERE KANKERSOORTEN:
Tobacco smoking and cancer: a brief review of recent epidemiological evidence.
This report summarises the epidemiological evidence on the association between tobacco smoking and cancer, which was reviewed by an international group of scientists convened by IARC. Studies published since the 1986 IARC Monograph on "Tobacco smoking" provide sufficient evidence to establish a causal association between cigarette smoking and cancer of the nasal cavities and paranasal sinuses, nasopharynx, stomach, liver, kidney (renal cell carcinoma) and uterine cervix, and for adenocarcinoma of the oesophagus and myeloid leukaemia. These sites add to the previously established list of cancers causally associated with cigarette smoking, namely cancer of the lung, oral cavity, pharynx, larynx, oesophagus, pancreas, urinary bladder and renal pelvis. Other forms of tobacco smoking, such as cigars, pipes and bidis, also increase risk for cancer, including cancer of the lung and parts of the upper aerodigestive tract. A meta-analysis of over 50 studies on involuntary smoking among never smokers showed a consistent and statistically significant association between exposure to environmental tobacco smoke and lung cancer risk. Smoking is currently responsible for a third of all cancer deaths in many Western countries. It has been estimated that every other smoker will be killed by tobacco.
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Big'r
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het duurde maar liefst 46 posts alvorens iemand de bewering van roxxe wetenschappelijk weerlegde.
Karma voor big'r
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Heb je mijn link niet gelezen Thinky
Post nr 4 of 5 geloof ik.en en in de eerste regel staat er in jouw artikel;
"Yes, it is true, smoking does not cause lung cancer. It is only one of many risk factors for lung cancer"
en iets verder:
"If they would say that smoking increases the incidence of lung cancer or that smoking is a risk factor in the development of lung cancer, then I would agree."
Heb je mijn link niet gelezen Thinky
ah ja net gezien, sorry, I stand corrected!
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Roxxe houdt zichzelf graag voor de gek.
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Net alsof ik de moeite ga doen om een tekst te zoeken waarin staat wat iedereen al weet.
justlooking
Dutch Bodybuilder
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lol weer die hypocrisie
wietgebruikers zijn slachtoffers, ze kunnen toch wel depressies en dergelijke krijgen
en sigaretterokers zijn des duivels
pff crazy belgie
Huh??
Ik zei dat mensen die tegen het rookverbod zijn met twee maten lopen te meten. Ze vinden dat mensen die zeggen dat het stinkt niet moet zeuren omdat dat nu eenmaal bij uitgaan hoort, maar zelf vinden ze wiet en dergelijke vieze, stinkende troep.
ja dat begrijp ik maar het is altijd beter een gefundeerd antwoord te geven dan te zeggen: "dat weet toch iedereen".
Big'r
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Geloof wat je wilt. Als je wilt aantonen dat niet 100% van de rokers longkanker krijgt, dan was dat al duidelijk.
De kans op longkanker ligt echter wel veel hoger. Ook volgens je eigen stuk. 800% vergeleken met een nietroker:
Maar je hebt gelijk roxxe, er ligt teveel nadruk op het gevaar van roken voor longkanker terwijl de kans op een veelheid aan andere kankersoorten ook stijgt. De kans om te overlijden bij hartziekten daalt met 36% voor mensen die zijn gestopt met roken vergeleken met rokers.
Tobacco smoking and cancer: a brief review of recent epidemiological evidence.
This report summarises the epidemiological evidence on the association between tobacco smoking and cancer, which was reviewed by an international group of scientists convened by IARC. Studies published since the 1986 IARC Monograph on "Tobacco smoking" provide sufficient evidence to establish a causal association between cigarette smoking and cancer of the nasal cavities and paranasal sinuses, nasopharynx, stomach, liver, kidney (renal cell carcinoma) and uterine cervix, and for adenocarcinoma of the oesophagus and myeloid leukaemia. These sites add to the previously established list of cancers causally associated with cigarette smoking, namely cancer of the lung, oral cavity, pharynx, larynx, oesophagus, pancreas, urinary bladder and renal pelvis. Other forms of tobacco smoking, such as cigars, pipes and bidis, also increase risk for cancer, including cancer of the lung and parts of the upper aerodigestive tract. A meta-analysis of over 50 studies on involuntary smoking among never smokers showed a consistent and statistically significant association between exposure to environmental tobacco smoke and lung cancer risk. Smoking is currently responsible for a third of all cancer deaths in many Western countries. It has been estimated that every other smoker will be killed by tobacco.
Smoking cessation for the secondary prevention of coronary heart disease.
BACKGROUND: Although the importance of smoking as a risk factor for coronary heart disease is beyond doubt, the speed and magnitude of risk reduction when a smoker with coronary heart disease quits are still subjects of debate. OBJECTIVES: To estimate the magnitude of risk reduction when a patient with CHD stops smoking. SEARCH STRATEGY: We searched the Cochrane Register of Controlled Trials (CENTRAL), MEDLINE, EMBASE, Science Citation Index, CINAHL, PsychLit, Dissertation Abstracts, BIDS ISI Index to Scientific and Technical Proceedings, UK National Research Register from the start of each database. Sixty-one large international cohort studies of cardiovascular disease were identified, and contact made with authors to search for any unpublished results. The search was supplemented by cross-checking references and contact with various experts. Date of last search was April 2003. SELECTION CRITERIA: Any prospective cohort studies of patients with a diagnosis of CHD, which include all-cause mortality as an outcome measure. Smoking status must be measured on at least two occasions to ascertain which smokers have quit, and followed-up for at least two years. DATA COLLECTION AND ANALYSIS: Eligibility and trial quality were assessed independently by two reviewers. MAIN RESULTS: Twenty studies were included. There was a 36% reduction in crude relative risk (RR) of mortality for those who quit smoking compared with those who continued to smoke (RR 0.64, 95% confidence interval 0.58 to 0.71). There was also a reduction in non-fatal myocardial infarctions (crude RR 0.68, 95% confidence interval 0.57 to 0.82). Many studies did not adequately address quality issues, such as control of confounding, and misclassification of smoking status. However, there was little difference in the results for the six 'higher quality' studies, and little heterogeneity between these studies. This review was not able to assess how quickly the risk of mortality was reduced. REVIEWER'S CONCLUSIONS: Quitting smoking is associated with a substantial reduction in risk of all-cause mortality among patients with CHD. The pooled crude RR was 0.64 (95% CI 0.58 to 0.71). This 36% risk reduction appears substantial compared with other secondary preventive therapies such as cholesterol lowering which have received greater attention in recent years. The risk reduction associated with quitting smoking seems consistent regardless of differences between the studies in terms of index cardiac events, age, coïtus, country, and time period. However, relatively few studies have included large numbers of older people, women, or people of non-European descent, and most were carried out in Western countries.
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Ik hoop niet dat DAT zijn motief is, maar dat zal vast niet. Wie verder denkt dan zijn neus lang is, weet dat een verbod in openbare gebouwen, horeca, .. het meeste effect heeft op de rokers zelf en de rokers eigenlijk genoodzaakt worden niet meer te roken.. of toch ten minste minder..