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Invloed anabolen op intelligentie
- Topic starter bellafonte
- Startdatum
- Recente activiteit Recente activiteit:
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BigMaus zei:Ik zeg tegen iemand laat het rusten...blijk van goede wil...maar ook die post is ineens weg
Omdat je in jouw post een 'slechte' post gequote had.
Lauw zei:ik poste ook iets heel nuttigs, en het is ook wegt zal de warmte wel zijn...
Jij postte niets nuttigs.
BigMaus zei:
Idd, het is 1 groot complot hier om mensen ervan te weerhouden goede posts te plaatsen enzo.
Lauw zei:ja man. Waar moet het heen als je niet meer anoniem vanachter je pc mensen op het internet kan beledigen?
http://forum.fok.nl
Nu weer svp ontopic? Blijf het niet lief vragen namelijk. Opmerkingen over deze post via PM/mail/MSN/postduif ofzo, maar niet hieronder.
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MrJ zei:Omdat je in jouw post een 'slechte' post gequote had.
Jij postte niets nuttigs.
Idd, het is 1 groot complot hier om mensen ervan te weerhouden goede posts te plaatsen enzo.
http://forum.fok.nl
Nu weer svp ontopic? Blijf het niet lief vragen namelijk. Opmerkingen over deze post via PM/mail/MSN/postduif ofzo, maar niet hieronder.
Invloed anabolen op intelligentie
Zie hier het levende bewijs...
Laat ik in het midden of het positief of negatief is
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hitman zei:Het is net politiek jongens dat heen en weer .
Wacht effe .. zou je denken ... nee... of zit onze ministers ook aan de roids.
Oke oke nu weer ontopic, of aas je intelligentie beinvloed. uhh wat wou ik ook al weer zeggen?
Jou hersenen zijn inmiddels al grotendeels aangetast aan je gedrag te zien de laatste tijd
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Infinity zei:Jou hersenen zijn inmiddels al grotendeels aangetast aan je gedrag te zien de laatste tijd
Ik zal het niet ontkennen, ik gedraag me de laatste tijd ook heel netjes, zo ken ik mezelf helemaal niet man
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ben er van overtuigd dat roids iedergeval je heldere momenten/concentratie voor een bepaalde periode kan tegengaan...hoog/laag t3, dopamine, test en nog 1 miljoen andere hormonen hebben als bijwerking oa concentratie verlies en niet helder kunnen denken(bromocriptine)...
hoeft niet te gebeuren maar kan wel..
hoeft niet te gebeuren maar kan wel..
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weenie...zijn bv ook van die mensen die niet genoeg t3/t4 produceren en met supplementatie van een van die twee worden ze gelijk stuk energieker/geconcentreeerder/helder. Modafinil schijnt trouwens wel goeie shit te zijn voor de heldere momenten...zal keertje die shit proberen misschien kan ik dan iets nuttigs posten...maar reken daar maar niet op..MrJ zei:Maar waarom kan het dan ook niet de andere kant op?
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Modafinil? interesting..
"...modafinil ('Provigil', 'Alertec', 'Vigicer', 'Modalert', etc) is a memory-improving and mood-brightening psychostimulant. It enhances wakefulness and vigilance, but its pharmacological profile is notably different from the amphetamines, methylphenidate (Ritalin) or cocaine. Modafinil is less likely to cause jitteriness, anxiety, or excess locomotor activity - or lead to a hypersomnolent 'rebound effect' - than traditional stimulants. Subjectively, it feels smoother and cleaner than the amphetamines too. It may even be anxiolytic. The normal elimination half-life of modafinil in humans is between 12 - 15 hours. So it's worth fine-tuning one's dosage schedule accordingly.
Current research suggests modafinil, like its older and better-tested analogue adrafinil, is a safe, effective and well-tolerated agent. It is long-acting and doesn't tend to cause peripheral sympathetic stimulation. Yet its CNS action isn't fully understood. Modafinil induces wakefulness in part by its action in the anterior hypothalamus. Its dopamine-releasing action in the nucleus accumbens is weak and dose-dependent; the likelihood of a euphoric response ('abuse potential'), dose-escalation and tolerance is thus apparently small. Modafinil has central alpha 1-adrenergic agonist effects i.e. it directly stimulates the receptors. Modafinil inhibits the reuptake of noradrenaline by the noradrenergic terminals on sleep-promoting neurons of ventrolateral preoptic nucleus (VLPO). More significant, perhaps, is its ability to increase excitatory glutamatergic transmission. This reduces local GABAergic transmission, thereby diminishing GABA(A) receptor signalling on the mesolimbic dopamine terminals.
Modafinil is proving clinically useful in the treatment of narcolepsy, a neurological disorder marked by uncontrollable attacks of daytime sleepiness. Narcolepsy is caused by dysfunction of a family of wakefulness-promoting and sleep-suppressing peptides, the orexins. Orexin neurons are activated by modafinil. Orexinergic neurons are found exclusively in the lateral hypothalamic area. Their activation is associated with enhanced pleasure-seeking and motivation as well as arousal. Orexinergic fibers project to the entire central nervous system. Genetically modified orexin-knockout animals offer a model of human narcolepsy. Narcoleptics suffer profound disturbances in normal sleeping patterns and variable degrees of depression. These symptoms can be reversed with modafinil. Selective orexin receptor agonists of the future may prove useful both to narcoleptics and the population at large.
Experimentally, modafinil is also used in the treatment of Alzheimer's disease, depression, attention-deficit disorder (ADHD), myotonic dystrophy, multiple sclerosis-induced fatigue, post-anaesthesia grogginess, cognitive impairment in schizophrenia, spasticity associated with cerebral palsy, age-related memory decline, idiopathic hypersomnia, jet-lag, and everyday cat-napping. Depressives who feel sleepy and fatigued on SSRIs can augment their regimen with modafinil. In September 2003, an advisory panel to the FDA endorsed its use for treating shift work sleep disorder and obstructive sleep apnea.
The US military are interested in modafinil too.
Modafanil is marketed as 'Alertec" in Canada - and over the Net. 'Alertec' is less expensive than 'Provigil'. Cheap generic modafinil has been available since 2006. But Cephalon is vigorously litigating to defend its patents.
In March 2005, Cephalon filed a New Drug Application (NDA) with the FDA for 'Nuvigil' (r-modafinil, armodafinil) - a single isomer formulation of modafinil. Nuvigil will be marketed aggressively to offset the anticipated loss of revenue from Provigil.
Modafinil is increasingly used as a 'lifestyle drug' - a lucrative 'off-label' market its makers have not been unduly keen to discourage. Some prescribing physicians have reportedly been surprised at a previously hidden epidemic of narcolepsy among hard-working young professionals attending their surgeries.
Prudence, however, should be exercised in drastically curtailing one's sleep. Prolonged sleeplessness weakens immune function. Animals tortured in sleep-deprivation experiments eventually die from massive bacterial infections of the blood..."
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"...modafinil ('Provigil', 'Alertec', 'Vigicer', 'Modalert', etc) is a memory-improving and mood-brightening psychostimulant. It enhances wakefulness and vigilance, but its pharmacological profile is notably different from the amphetamines, methylphenidate (Ritalin) or cocaine. Modafinil is less likely to cause jitteriness, anxiety, or excess locomotor activity - or lead to a hypersomnolent 'rebound effect' - than traditional stimulants. Subjectively, it feels smoother and cleaner than the amphetamines too. It may even be anxiolytic. The normal elimination half-life of modafinil in humans is between 12 - 15 hours. So it's worth fine-tuning one's dosage schedule accordingly.
Current research suggests modafinil, like its older and better-tested analogue adrafinil, is a safe, effective and well-tolerated agent. It is long-acting and doesn't tend to cause peripheral sympathetic stimulation. Yet its CNS action isn't fully understood. Modafinil induces wakefulness in part by its action in the anterior hypothalamus. Its dopamine-releasing action in the nucleus accumbens is weak and dose-dependent; the likelihood of a euphoric response ('abuse potential'), dose-escalation and tolerance is thus apparently small. Modafinil has central alpha 1-adrenergic agonist effects i.e. it directly stimulates the receptors. Modafinil inhibits the reuptake of noradrenaline by the noradrenergic terminals on sleep-promoting neurons of ventrolateral preoptic nucleus (VLPO). More significant, perhaps, is its ability to increase excitatory glutamatergic transmission. This reduces local GABAergic transmission, thereby diminishing GABA(A) receptor signalling on the mesolimbic dopamine terminals.
Modafinil is proving clinically useful in the treatment of narcolepsy, a neurological disorder marked by uncontrollable attacks of daytime sleepiness. Narcolepsy is caused by dysfunction of a family of wakefulness-promoting and sleep-suppressing peptides, the orexins. Orexin neurons are activated by modafinil. Orexinergic neurons are found exclusively in the lateral hypothalamic area. Their activation is associated with enhanced pleasure-seeking and motivation as well as arousal. Orexinergic fibers project to the entire central nervous system. Genetically modified orexin-knockout animals offer a model of human narcolepsy. Narcoleptics suffer profound disturbances in normal sleeping patterns and variable degrees of depression. These symptoms can be reversed with modafinil. Selective orexin receptor agonists of the future may prove useful both to narcoleptics and the population at large.
Experimentally, modafinil is also used in the treatment of Alzheimer's disease, depression, attention-deficit disorder (ADHD), myotonic dystrophy, multiple sclerosis-induced fatigue, post-anaesthesia grogginess, cognitive impairment in schizophrenia, spasticity associated with cerebral palsy, age-related memory decline, idiopathic hypersomnia, jet-lag, and everyday cat-napping. Depressives who feel sleepy and fatigued on SSRIs can augment their regimen with modafinil. In September 2003, an advisory panel to the FDA endorsed its use for treating shift work sleep disorder and obstructive sleep apnea.
The US military are interested in modafinil too.
Modafanil is marketed as 'Alertec" in Canada - and over the Net. 'Alertec' is less expensive than 'Provigil'. Cheap generic modafinil has been available since 2006. But Cephalon is vigorously litigating to defend its patents.
In March 2005, Cephalon filed a New Drug Application (NDA) with the FDA for 'Nuvigil' (r-modafinil, armodafinil) - a single isomer formulation of modafinil. Nuvigil will be marketed aggressively to offset the anticipated loss of revenue from Provigil.
Modafinil is increasingly used as a 'lifestyle drug' - a lucrative 'off-label' market its makers have not been unduly keen to discourage. Some prescribing physicians have reportedly been surprised at a previously hidden epidemic of narcolepsy among hard-working young professionals attending their surgeries.
Prudence, however, should be exercised in drastically curtailing one's sleep. Prolonged sleeplessness weakens immune function. Animals tortured in sleep-deprivation experiments eventually die from massive bacterial infections of the blood..."
ff aan me supplier vragen
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Test beïnvloed hersenfuncties op een positieve manier: betere concentratie; focus, assertiviteit (die bij sommige individuen kan omslaan - of doorslaan- in agressie), en alghele mentale "scherpte".
Ik ken iemand die van dit gegeven gebruik maakte en zijn kuurtjes zo plande dat hij in een belangrijke tentamenweek "on cycle" was.
Wat verder alle psychische (bij)werkingen zijn, weet ik niet, maar ik denk dat er heel veel bullshit-onderzoeken de rondte gaan....
Ik ken iemand die van dit gegeven gebruik maakte en zijn kuurtjes zo plande dat hij in een belangrijke tentamenweek "on cycle" was.
Wat verder alle psychische (bij)werkingen zijn, weet ik niet, maar ik denk dat er heel veel bullshit-onderzoeken de rondte gaan....
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lol... ja gebruik in mate is misschien wel de oplossingECLIPSE zei:het kan idd omslaan naar een soort tijdelijke paranoia ja klaas
...